help button home button Endocrine Society JCEM
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS

Journal of Clinical Endocrinology & Metabolism Vol. 50, No. 1 1-4
doi:10.1210/jcem-50-1-1
Copyright © 1980 by the Endocrine Society.
This Article
Right arrow Full Text (PDF)
Right arrow Submit a related Letter to the Editor
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow Request Copyright Permission
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by BECK-NIELSEN, H.
Right arrow Articles by PEDERSEN, O.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by BECK-NIELSEN, H.
Right arrow Articles by PEDERSEN, O.

Prednisone Increases the Number of Insulin Receptors on Monocytes from Normal Subjects

HENNING BECK-NIELSEN, ROBERTO DE PIRRO* and OLUF PEDERSEN

Medical Department III and Department of Clinical Chemistry, County Hospital Aarhus, 8000 C, Denmark

Address requests for reprints to: Dr. H. Beck-Nielsen, Medical Department III, Amtssygehuset, Tage Hansensgade, 8000 Aarhus, C, Denmark.

The effect of prednisone on insulin receptors on circulating monocytes was studied in 38 normal volunteers. Intake of prednisone in doses which are usually employed in clinical treatment (40 mg/day) was associated with a significant increase in the number of insulin receptors. The rise in insulin binding was maximal 7 h after the beginning of the medication and it was dose related.

* Present address: 2' Clinica Medica, Policlinico Umberto 10, Roma, Italy.

Received March 6, 1979.




This article has been cited by other articles:


Home page
DiabetesHome page
S. A. Summers and D. H. Nelson
A Role for Sphingolipids in Producing the Common Features of Type 2 Diabetes, Metabolic Syndrome X, and Cushing's Syndrome
Diabetes, March 1, 2005; 54(3): 591 - 602.
[Abstract] [Full Text] [PDF]


Home page
Am. J. Physiol. Endocrinol. Metab.Home page
M. F. Nielsen, A. Caumo, V. Chandramouli, W. C. Schumann, C. Cobelli, B. R. Landau, H. Vilstrup, R. A. Rizza, and O. Schmitz
Impaired basal glucose effectiveness but unaltered fasting glucose release and gluconeogenesis during short-term hypercortisolemia in healthy subjects
Am J Physiol Endocrinol Metab, January 1, 2004; 286(1): E102 - E110.
[Abstract] [Full Text]


Home page
ANN INTERN MEDHome page
S. A. KAPLAN, B. M. LIPPE, C. R. BRINKMAN III, M. B. DAVIDSON, and M. E. GEFFNER
Diabetes Mellitus
Ann Intern Med, May 1, 1982; 96(5): 635 - 649.
[Abstract] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Endocrinology Endocrine Reviews J. Clin. End. & Metab.
Molecular Endocrinology Recent Prog. Horm. Res. All Endocrine Journals
Copyright © 1980 by The Endocrine Society